DISCUSSION: Urica™ consists of an advanced blend of compounds designed to help support healthy uric acid levels.*
 

AOR guarantees that no ingredients not listed on the label have been added to the product. Contains no corn, wheat, gluten, nuts, dairy, soy, eggs, fish or shellfish.

Suggested Use
Take 4 capsules daily, or as directed by a qualified health care professional.

Source
Multi-Sourced – various herb and plant sources

Pregnancy / Nursing
Do not take.

Cautions
Consume with at least one litre of water throughout the day.

ADDITIONAL INFORMATION / RESEARCH:

Gout, also referred to as metabolic arthritis or acute inflammatory monoarthritis, is caused by an abnormal build-up of uric acid in the blood - uric acid being a metabolic by-product of purine metabolism. Purines are natural substances found in many foods, and particularly in high protein foods like organ meats and fish like mackerel and sardines. Excessive blood levels of uric acid then lead to acute inflammation of the joints. 'Classic Gout' which usually targets the joints of the big toe and other joints within the legs and feet and can affect people of all ages. However, another type of gout - called 'Atypical Gout' - can affect any joint in either the arms or legs and is observed predominantly in the elderly. Both types of gout are characterized by excruciating pain and swelling. In many typical cases of gout, patients are overweight, predisposed to Type II diabetes and hypertension, and are at a higher risk of cardiovascular disease. Gout is disproportionately found in societies whose diets include large amounts of protein, fat and alcohol. Since a fundamental facet of gout is the metabolism of protein, the areas of kidney, bladder and urinary tract health are primary targets for treatment.

References

Yu Z, et al. Morin (3,5,7,2',4'-pentahydroxyflavone) exhibits potent inhibitory actions on urate transport by the human urate anion transporter (hURAT1) expressed in human embryonic kidney cells. Drug Metab Dispos. 2007 Jun;35(6):981-6.

Yu Z, et al. The dual actions of morin (3,5,7,2',4'-pentahydroxyflavone) as a hypouricemic agent: uricosuric effect and xanthine oxidase inhibitory activity. J Pharmacol Exp Ther. 2006 Jan;316(1):169-75.

Singh A, et al. An experimental evaluation of possible teratogenic potential in Boerhaavia diffusa in Albino rats. Planta Med. 1991 Aug;57(4):315-6.

Mudgal V. Studies on medicinal properties of Convolvulus pluricaulis and Boerhaavia diffusa. Planta Med. 1975 Aug;28(1):62-8.

Anand R, et al. Activity of certain fractions of Tribulus terrestris fruits against experimentally induced urolithiasis in rats. Indian J Exp Biol. 1994 Aug;32(8):548-52.

Sannd BN, Kumar A, Kumar N. J.R.A.S., Vol. XIV, No., 3-4, pp. 98-114.

Abd-El-Malek YA, et al. Antimicrobial principles in leaves of Lawsonia inermis L. Zentralbl Bakteriol Parasitenkd Infektionskr Hyg. 1973;128(1):61-7.

Ballabh B et al. Traditional medicinal plants of cold desert Ladakh-Used against kidney and urinary disorders. J Ethnopharmacol. 2008 Jul 23;118(2):331-9.

Joshi VS, et al. Inhibition of the growth of urinary calcium hydrogen phosphate dihydrate crystals with aqueous extracts of Tribulus terrestris and Bergenia ligulata. Urol Res. 2005 May;33(2):80-6.

Umamaheswari M, et al. Xanthine oxidase inhibitory activity of some Indian medical plants. J Ethnopharmacol. 2007 Feb 12;109(3):547-51.

Lin JK, Shih CA. Inhibitory effect of curcumin on xanthine dehydrogenase/oxidase induced by phorbol-12-myristate-13-acetate in NIH3T3 cells. Carcinogenesis. 1994 Aug;15(8):1717-21.

Jia Z, et al. EPR studies on the superoxide-scavenging capacity of the nutraceutical resveratrol. Mol Cell Biochem. 2008 Jun;313(1-2):187-94.

Du Y, et al. Grape seed polyphenols protect cardiac cells from apoptosis via induction of endogenous antioxidant enzymes. J Agric Food Chem. 2007 Mar 7;55(5):1695-701.
 

 *These statements have not been evaluated by the Food and Drug Administration. This product is not intended to diagnose, treat, cure, or prevent any disease.

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